Longevity ⏱ Half-life: Plasma half-life is short, but cellular NAD+ pool restoration has prolonged metabolic effects.

NAD+

Nicotinamide Adenine Dinucleotide

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Half-Life
Plasma half-life is short, but cellular NAD+ pool restoration has prolonged metabolic effects.
Mol. Weight
663.43 g/mol

What is NAD+?

NAD+ is a critical coenzyme present in all living cells. While not a peptide, it is a cornerstone compound in longevity research and is commonly available through research peptide suppliers. Its role in cellular energy production, DNA repair, and epigenetic regulation places it at the center of aging biology.

Research Applications

Aging and longevity research, sirtuin biology, mitochondrial function, DNA repair, neurodegenerative disease models, and metabolic health.

Dosage Information (Research Use)

Research administration routes: subcutaneous, intramuscular, and intravenous at various concentrations. Oral precursors (NMN, NR) also widely studied. Research use only.

Reconstitution & Handling

Dissolve in sterile water or bacteriostatic water. NAD+ is sensitive to light and heat — prepare fresh and use promptly.

Half-Life & Pharmacokinetics

Plasma half-life is short, but cellular NAD+ pool restoration has prolonged metabolic effects.

Reported Observations in Literature

IV administration: flushing, nausea, cramping (generally transient). Subcutaneous: injection site discomfort. Oral precursors generally well-tolerated.

Key Research References

  • Yoshino J, et al. “NAD+ intermediates: The biology and therapeutic potential of NMN and NR.” Cell Metab. 2018

How NAD+ Works

NAD+ (Nicotinamide Adenine Dinucleotide) is an essential coenzyme found in every cell, participating in over 500 enzymatic reactions. While technically not a peptide, it is commonly supplied by peptide research vendors due to overlapping research applications. Primary mechanisms include serving as an electron carrier in mitochondrial energy production, activating sirtuin enzymes (SIRT1-7) involved in DNA repair and longevity pathways, supporting PARP-mediated DNA repair, and maintaining cellular redox balance.

Research Findings

NAD+ levels decline approximately 50% between ages 40 and 60 in human tissue studies. This decline correlates with age-related metabolic dysfunction, mitochondrial dysfunction, and impaired DNA repair. Subcutaneous and IV administration bypass the bioavailability challenges of oral supplementation.

Dosage & Administration

Research administration routes: subcutaneous, intramuscular, and intravenous at various concentrations. Oral precursors (NMN, NR) also widely studied. Research use only.

Safety & Side Effects

IV administration: flushing, nausea, cramping (generally transient). Subcutaneous: injection site discomfort. Oral precursors generally well-tolerated.

Important: All safety information is derived from published research, primarily animal studies. No controlled human clinical trial data exists unless explicitly noted. This compound is sold for research purposes only.

Quick Facts

Sequence Non-peptide (dinucleotide coenzyme)
Molecular Weight 663.43 g/mol
Half-Life Plasma half-life is short, but cellular NAD+ pool restoration has prolonged metabolic effects.
Available Sizes 500mg, 1000mg
Storage Lyophilized: -20°C. Reconstituted: 2-8°C, use promptly.

Key Research References

  • Yoshino J, et al. "NAD+ intermediates: The biology and therapeutic potential of NMN and NR." Cell Metab. 2018

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